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BPC-157’s research base is anchored in the work of Sikiric and colleagues at the University of Zagreb, who have published extensively on the peptide since the early 1990s

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Considering that apoptosis contributes minimally to human heart failure, with only 80–250 cardiomyocytes per 100,000 affected, non-apoptotic cell death mechanisms, including ferroptosis, gain prominence in this context [12, 13]