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Studies have shown that when amyloid-beta and tau are increased, the cell’s ability to neutralize oxidative stress and mitochondria specific ROS is decreased leading to further tau hyperphosphorylation and neurodegeneration ( These redox imbalances also impact astrocytes, which normally synthesize GSH through the consecutive sections of two ATP consuming cytosolic enzymes

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In the presence of Dopachrome Tautomerase (DCT/TYRP2) activity, DC is converted into DHICA